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Metabolic & Cardiometabolic · Gut & Microbiome

Do gut-bacteria bile acids boost your appetite hormone?

The claim, precisely: secondary bile acids stimulates GLP-1

Strong support Metabolic & Cardiometabolic 🐭 Non-human evidence
RefutedContestedStrong support
consensus score 1.00

Yes as a mechanism, but only shown in animals so far — human evidence is correlational.

Evidence ladder

How far up the ladder this claim has climbed. A high consensus on a low rung means "consistent so far," not "proven in people."

Top evidence so far: Animal studies (Animal)

MechanismIn-vitroAnimalObservationalRCTMeta-analysis

How the studies fall

6 support 0 contradict 0 tested null 0 mixed · 3 sources, 3 independent groups

What the evidence shows

Microbe-generated secondary bile acids (DCA/LCA, taurine-conjugated) activate basolateral TGR5/GPBAR1 on L-cells to stimulate GLP-1 — robust in animal/ex-vivo models; the diet->microbe->secondary-BA->GLP-1 chain is causal in mice, human evidence is correlational only.

The evidence (6)

SourceGradeStanceQualityFinding
Zheng X, et al.
2021 · (HCA)
animal supports moderate [FT-verified] Zheng 2021 CellMetab hyocholic acids via TGR5+FXR mouse+human corr. ANIMAL/MECHANISM
Zheng X, et al.
2021 · (HCA)
animal supports moderate Hyocholic acid species dual TGR5-agonist/FXR-antagonist raise GLP-1; low serum HCA assoc. with human diabetes
Pathak P, et al.
2018 · (FXR/TauroLCA)
animal supports moderate Intestinal FXR agonist + microbiota -> tauroLCA -> TGR5 -> GLP-1; antibiotics reversed it
Pathak P, et al.
2018 · (FXR/TauroLCA)
animal supports moderate [FT-verified] Pathak/Chiang 2018 mouse microbiota+FXR activate TGR5 to improve metabolism. ANIMAL-only
Brighton CA, et al.
2015 · Endocrinology
mechanism supports moderate [FT-verified] Brighton/Gribble 2015 murine/ex-vivo L-cell BA->GLP-1 via basolateral TGR5. ANIMAL/MECHANISM-only
Brighton CA, et al.
2015 · Endocrinology
animal supports high TDCA>TCA via basolateral GPBAR1; abolished in GPBAR1-KO; luminal needs ASBT

Educational only, not medical advice. Grades and scores reflect published evidence weighted by study design and quality; see the methodology.